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Smoking habit in patients with recurrent aphthous stomatitis
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Smoking habit in patients with recurrent aphthous stomatitis (RAS)
Laura Fernández 2º AP
WHAT IS IT?
Common disease in which painful and recurrent round or oval ulcers appear on the oral mucosa. It is very common but the etiology is not clear.
Tobacco consumption is related to this pathology; it exerts a protective effect whose origin is not understood. Therefore this article investigates different possible reasons.
- Most patients with RAS are non-smokers and there is a lower prevalence of RAS among smoking patients.
- In many patients, the cessation of this habit is related to the appearance of new lesions, a worsening of the disease or even the onset of the disease.
STUDIO 1:
Data was collected on the tobacco consumption in 171 patients with this disease, 94 women and 77 men with a mean age of 43 years old.
In all cases so a clinical follow-up of the patient was carried out for a minimum of 12 months.
- 32 patients were ex-smokers and suffered an increase in injuries after stopping tobacco use.
- Furthermore, 5 of them the beginning of the illness coincided with the cessation of consumption.
- 16 patients were current smokers and had fewer injuries than the rest.
STUDIO 2:
The protective effect that tobacco exerts in patients with RAS may be due to the hyperkeratinization it causes in the oral mucosa. It would protect her from trauma.
STUDIO 3:
- Grady studied that smokeless tobacco and nicotine-containing lozenges appear to have the same effect, although it is not clear how.
- Cherubini has studied thiocyanate, component
specifically tobacco, in case it was the reason for the protective effect of tobacco on RAS. When comparing their concentration
salivary in patients with RAS, a clear relationship has not been established between their salivary levels and the prevalence of lesions
CONCLUSION:
We can observe from the results on the relationship between tobacco consumption and typology of lesions in patients with RAS and the data contributed by the works of other authors that the effect of tobacco on RAS could be a dose-dependent relationship.