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Oxidative Stress Heart Disease

Four studies already published at the National Institute of Health

Jacqueline Seavy King

on 12 December 2013

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Transcript of Oxidative Stress Heart Disease

These studies were completed and independently funded by the National Institute of Health, the American Heart Association and many other Universities and Medical Groups around the world!"
For more information or copy of complete study : 619-232-9229 or
Call for Copy of Complete Study
Virginia Commonwealth University
Medical Center

Chronic Pulmonary Artery Pressure Elevation is Insufficient to Explain Right Heart Failure.
“The animals pre-treated with Protandim experienced strong cardio-protective effects.

*"Protandim protected the animal's hearts by increasing the expression of protective genes and preventing the formation of scar tissue.”
Ohio State University

Protandim Attenuates Intimal Hyperplasia in Human Saphenous Veins Cultured Ex Vivo A Catalase Dependent Pathway
This study examined the biochemical mechanisms that underlie the ability of Protandim to suppress intimal hyperplasia-over-proliferation of cells that line the vessel wall-a common adverse event that limits the effectiveness of vascular surgery. Treatment of human saphenous veins with Protandim blocked intimal hyperplasia and reduced cellular proliferation to that of freshly isolated human saphenous veins.

*Protandim significantly increased antioxidant enzyme activity in veins while reducing free radical levels, lipid peroxidation and intimal proliferation.
Colorado State University

Phytochemical Activation of Nrf2 Protects Human Coronary Artery Endothelial Cells against an Oxidative Challenge.
“These results show Protandim induces Nrf2 nuclear localization and antioxidant enzyme expression, and protection of Human coronary artery endothelial cells from oxidative challenge is Nrf2 dependent. “
Virginia Commonwealth University Medical Center

Antioxidants for
the treatment of
patients with severe angioproliferative
“Treatment of Su/Hx rats with Protandim which Nrf2-dependently up-regulates the expression of genes encoding antioxidant enzymes, protects against right heart failure without affecting angiobliteratiove PAH.”
A clip from the Triple bypass & stint surgery study and Protandim follows in this playlist.
Next watch a clip from the Triple bypass & stint surgery study.
Call for Copy of Complete Study
November 17, 2009
Call for Copy of Complete Study
March 15, 2011
May 22, 2012
August 7, 2012
Full transcript