Management of obesity_Juthi Saha

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by JUTHI Saha18 views·Updated May 20, 2024

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  1. Good morning, students. I'm Juhi
  2. Shaha,
  3. faculty member of
  4. Khan Chara
  5. Mohala
  6. Nish Mohamed
  7. Daa.
  8. Today I'm going to discuss about management of obesity.
  9. According to World Health Organisation,
  10. in 2022 1 in eight people in the world were living with obesity.
  11. And the prevalence of obesity in India is increasing faster than world
  12. average.
  13. So we
  14. should have a clear knowledge about obesity, its, uh,
  15. causes and how it affects our body and how we can manage, uh, obesity.
  16. So first, we will start with the definition of obesity.
  17. So what is obesity? Obesity is a generalised, uh, accumulation of excess body fat
  18. in our body, which leads to, um, weight gain more than 20% of desirable weight.
  19. This, uh,
  20. uh, excess accumulation of, uh,
  21. adipose tissue may occur around the waist or trunk or, uh,
  22. peripherally around the body.
  23. So now we will discuss about the classification of obesity.
  24. Obesity can be classified
  25. on the basis of BM. I grade. Uh uh, Grade one obesity. Um, the grade one obese the BM.
  26. I is, uh, between, uh, 30 to 35 4.9 KG per metre squared
  27. and in case of grade two obesity.
  28. Uh, the BM I, uh, range is between, uh, 35 to 39.9 KG per metre square.
  29. And the last one is the grade three obesity. It is also known as morbid obesity.
  30. Um uh uh.
  31. BM I of grade three obese is more than 40 KG per metre square
  32. on the basis of, uh, uh, distribution of adipose tissue.
  33. Uh, we can, uh, classify the obesity in two classes.
  34. First one is android obesity, and the second one is, uh, gynoid obesity.
  35. Anatomically adipose tissue is unequally distributed in
  36. varying proportion in the human body.
  37. When the distribution of adipose tissue is abnormally
  38. high surrounding the internal viscera of abdomen,
  39. it is referred to as abdominal or central obesity.
  40. Uh,
  41. it is also referred as apple
  42. shaped obesity.
  43. Uh, when the, uh, excess accumulation of adipose tissue occurs, uh,
  44. in the lower part of the body,
  45. it is known as gynoid obesity or peripheral obesity or pure
  46. shaped obesity.
  47. Uh, on the basis of age here, we can classify obesity in two classes juvenile
  48. onset obesity and adult onset obesity.
  49. Juvenile
  50. onset obesity occurs due to hyperplasia, and most rapidly,
  51. in the first few years of life.
  52. There is a marked increase in the adequate tissue cells.
  53. Thus it is referred as hyperplastic obesity
  54. fat cells. Once developed, it doesn't disappear.
  55. For this reason, fat Children are inclined to be fat adults.
  56. As many as 80% of obese Children will become obese adults
  57. in adult
  58. onset obesity. The size of the individual fat cell is greatly enhanced.
  59. Thus,
  60. hypertrophic individuals maintain weight loss better than hyperplastic one.
  61. Now we will discuss the aetiology of obesity.
  62. Actually, obesity is a multifactorial disease.
  63. It occurs due to various, uh, reasons. Uh, including genetics, uh, food habits.
  64. Uh, uh, consumption of, uh, it junk food it. Sorry.
  65. Excess consumption of junk food, family history, psychological factors,
  66. endocrine factor and urbanisation also, uh, causes or, uh,
  67. also associated with the occurrence of
  68. ation of, uh, obesity.
  69. Now we will discuss about the pathophysiology of obesity.
  70. Brian plays the central role in food regulation.
  71. Actually, there is there, uh, is, uh, two, centre.
  72. The one is hunger centre, and another one is satiety
  73. centre, situated in the hypothalamus.
  74. When we are hungry, the hunger centre
  75. provide, um, signals.
  76. And we, uh,
  77. have
  78. the arch to consume food
  79. And when I when we are consuming food,
  80. the, uh uh,
  81. That in turn,
  82. provides signals to the satiety
  83. centre and to stop the feeding behaviour. By this, the food. Mm.
  84. Sorry. By this process, the Foo, uh, food regulation in human body is maintained,
  85. uh, when the energy out intake is greater than the energy output, uh,
  86. then the person will be in in a positive energy balance,
  87. and this ultimately results in obesity.
  88. Now, we will discuss about the complications of obesity.
  89. Obesity causes, uh, sexual hormone imbalance. Uh uh, increased, uh, free
  90. fatty acid, uh, in our blood. And it also causes metabolic stress.
  91. As a result, uh, there may be hormone related dependent tumours, uh,
  92. insulin resistance.
  93. Uh, hyper,
  94. uh,
  95. hyperlipidemia. Um and it's also result in type two diabetes, uh,
  96. amitis
  97. and different cardiovascular, uh, diseases. Uh, and, uh, there is sleep apnea.
  98. Um, osteoarthritis, low back pain and shortness of breath may occur.
  99. So, um,
  100. if we want to manage the
  101. obesity, we need to assess first.
  102. So for this reason,
  103. there are different parameters that we can use to assess obesity.
  104. The first one is BM I,
  105. which is also known as platelet in this.
  106. It is a ratio of weight, uh, in KG to height in metres square.
  107. Uh, as we are, we have discussed about the, uh, different, Um uh,
  108. classic, uh, different types of obesity on the basis of, uh, PM I
  109. There are several other, uh, assessment tools that we can use. Uh, that is BRCA
  110. index caps,
  111. uh, corpulence
  112. index, uh, pal
  113. index with
  114. hip ratio and through
  115. meric, uh, to, uh, measurement like, uh, skin fold thickness.
  116. Uh, these, uh, uh, parameters
  117. helps to assess the,
  118. um, uh, obesity.
  119. Now we will discuss about the management of obesity
  120. With recent economic progress and urbanisation.
  121. Traditional healthy diets have been
  122. substituted by Western influence.
  123. Diet rich in a saturated fat, refined sugar and preserve
  124. Weight loss and weight maintenance require a
  125. long term reduction in calorie intake.
  126. Long-term adherence to a healthy eating pattern that is, uh,
  127. personalised to meet individual values
  128. and preferences while fulfilling nutritional
  129. needs and treatment goals is an important element of,
  130. uh, managing health and weight.
  131. Diet plays an important role in, um, management of obesity.
  132. The diet, uh, 30 principle, uh, or the should be low calorie, uh, normal protein.
  133. Uh, limited carbohydrate, high fibre, restricted fat and liberal, uh, food, Uh,
  134. with the, uh, normal vitamins and minerals, Uh, except, uh,
  135. sodium are recommended.
  136. All individuals, regardless of the body size or composition, uh,
  137. would benefit from, uh, adopting a healthy,
  138. well balanced eating pattern and engaging in regular physical activity.
  139. Aerobic activity, uh,
  140. for 30 to 60 minutes on most days of the week can lead to a small amount of, uh,
  141. small but significant amount of weight and fat loss.
  142. Uh,
  143. stress, uh, is a major, uh, reason for overeating.
  144. And, uh, uh, patient can, uh, reduce, uh, stress
  145. related over
  146. eating by learning how to implement methods other
  147. than eating to deal with or reduce stress.
  148. And some examples include, uh, deep, uh, muscle relaxation, meditation, yoga.
  149. Uh, and, uh, they must ask to include physical activity in their daily routine.
  150. Drug therapy may be used as a part of comprehensive weight loss programme,
  151. including diet therapy and physical exercise.
  152. For patient with BM.
  153. I, uh, of, uh, 30 KG per metre squared with no concomitant obesity
  154. related risk factors or, uh, diseases. The new drugs, uh, cyber
  155. Ramin and all these that combined with, uh
  156. faline
  157. provide helpful tool to access healthcare professionals, uh,
  158. manage the weight of the patient more successfully.
  159. So most successful programmes employ a multidisciplinary
  160. approach to weight loss with the hypothalamic
  161. diet, behaviour modification to change eating, behaviour, aerobic exercise,
  162. pharmacological therapy, social
  163. support,
  164. emphasis must be on maintenance of weight
  165. loss.
  166. And
  167. with this I'm concluding my lecture.
  168. Thank you.