RARE CASE OF LATE-ONSET

Hi.

My name is Jennifer and my presentation is on

the rare case of late onset Narcolepsy Type one

Here is just an overview of narcoleptic type one symptoms and how to categorize them

as well as my individual case study down below

is considered a lifelong neurological disease and

is characterized by excessive daytime sleepiness,

cataplexy

as well as disrupted nocturnal sleep.

This disease is caused by the loss

of hypocrite neurons in the lateral hypothalamus.

The disease,

it's suggested to have an autoimmune ideology.

The typical age of onset is the second decade

in another smaller peak in the 4th decade

and

Any cases after like considered 50 years or 70 years is considered rare.

You too.

Less likelihood of cases being recognized or even

known about

and out to the side is just a small diagram showing healthy sleep

and

narcoleptic sleeping where you can see that it's fragmented

cataplexy is caused by a loss of

the chemical hypocrite,

in which is the brains alert system that regulates sleep and wake

due to the hypothalamus being damaged or destroyed.

Attacks of cataplexy may occur.

Attacks are provoked by strong emotions such as laughter

and is determined by loss of muscle tones with regained consciousness.

Hypnotic logic, hallucinations, right?

They have no gogic hallucinations are diagnosed by multiple sleep latency test.

These hallucinations or illusions appear dreamlike

to the patient from smell visually,

physically and auditory lee

near sleep onset and what occurred from sleeping to awake

is known as

hypno pump. It

sleep paralysis, also known as antonia

occurs in rem sleep and a patient may feel as if

they are breathless or suffocating

with the inability to move or speak during transitions and sleep

and feeling as if someone is standing or sitting on their chest,

through the chest pressure,

feelings that one is outside of one's body,

another symptom

and narcolepsy type one includes

instantaneous firm,

which can be

seen as muscular paralysis.

Poor nocturnal sleep, which contributes to feelings of sleepiness upon waking,

quick onset of rem sleep causes inadequate rest and correlating mental issues,

lots of muscle

control due to

protective protective of rem sleep.

Here we have our specific case study

and our uniqueness of the study. Livelihood.

Medical history testing and treatment as well as our bibliography

and the patient is 69 years old, retired

and living with his wife

and

I was diagnosed with excessive daytime sleepiness

and no known triggers such as illness.

He was previously treated for high cholesterol,

chronic obstructive preliminary disease

or to hypertension

in history of deep vein.

The

goose's,

He drinks a couple of beers

and smokes about 20 cigarettes every day

In this study is unique due to the age of onset. He was 69 when

he was diagnosed with excessive daytime sleepiness. He had no

sleep onset rem detected in nocturnal sleep. In the first trials.

A neurological exam did not reveal

any neurological cognitive or physical findings,

no symptoms of sleep paralysis or initial signs of

I've gotten it

hallucinations until patient woke up to put imaginary dog from a dream.

The patient reported

That he lives with his wife. He's retired and likes to fish

in his free time. He likes to smoke approximately 20 cigarettes a day

and drinks a couple of beers. He also falls asleep watching TV reading

and on on an iPad versus doing physical things such as such as fishing

where he doesn't tend to fall asleep as much.

The patient's medical history includes

previous diagnosis with preliminary disease,

history of deep vein thrombosis,

arterial hypertension and a history of high cholesterol.

Through diagnostic testing, the

multiple sleep latency test showed

the sleep latency of 2.1 minutes

And the patient slept in five out of five studies achieved Raymond three tests

while the multiple sleep wait

in diagnostic testing.

The

maintenance of wakeful test

performed in order to renew his license

To drive

confirmed excessive daytime sleepiness.

And the patient slept in all four tests

With a sleep latency of

0.25 minutes.

Mhm.

The pain. The patient's treatment included a refusal of stimulant.

However,

the patient was also given

climate prime

Which started at 250 mg and over a few months was increased to 50 mg.

The patient was overall satisfied

with

the patient was overall satisfied

to cope with excessive daytime sleepiness by

napping after meals.

And this is my bibliography here,

the resources that I used and thank you for tuning in

RARE CASE OF LATE-ONSET

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