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Cause of heart disease is high homocysteine level

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by

AHMED SALEH

on 16 January 2014

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Transcript of Cause of heart disease is high homocysteine level

Cause
of
heart
is
High
disease
Homocysteine
level
70%
of
heart
Occur
attacks
in
people
with
little
or
no
previous
arterial
blockage.
70%
LDL
cholesterol
Only
becomes
bad
when
it
is
damaged
by
oxidative
stress
causing
cholesterol
to
become
sticky
thus
forming
plague
buildup
Studying
Children
who
died
of
a
heart
attack
at
very
early
Dr.
Kilmer
McCully
discovered
in
1969
homocysteine
to
be
the
underlying
cause.
ages
Homocysteine
is
second
only
to
cigarette
smoking
in
causing
Oxidative
damage
Cholesterol
Arterial
wall
Only
Nutrients
found
in
Vitamins
Minerals
Antioxidants
Have
been
scientifically
to
reduce
homocysteine.
shown
Normal
range
your
doctor
will
say
is
acceptable
is
5-15
micromoles
per
liter
of
blood.
LDL
cholesterol
oxidative
stress
Plague
buildup
No
known
man
made
drug
can
reduce
homocysteine
Precisely
Why
drug
companies
do
not
educate
doctors
on
homocysteine?
They
can
not
profit
from
it!
Hcy
But
Homocysteine
Research
Shows
Reducing
abnormally
high
levels
(above 7)
drastically
Can
Reduce
your
chances
of
heart
disease.
Any
3
point
increase
Equates
To
A
35
higher
risk
of
heart
attack
35%
%
Homocysteine
is
naturally
produced
in
the
body
As
amino
acid.
In
the
presence
of
sufficient
amounts
of
Folic
acid
Vitamin
B12
Vitamin
B6
Homocysteine
is
metabolically
synthesized
into
Cysteine
and
Methionine.
an
Folic
acid
Vitamin
B12
Vitamin
B6
Deficiencies
of
these
nutrients
Lead
to
Obstruction
of
this
process.
The
result
is
high
levels
of
homocysteine
in
the
blood
stream
Causing
widespread
damage
to
the
inner
shell
of
and
vessels
Oxidative
Damage
To
LDL
Cholesterol
Think
of
this
Oxidative
damage
like
a
prickly
pine
cone
lining
your
blood
vessels.
Plaque
now
has
a
much
higher
chance
of
sticky
and
arterial
blockage
Robinson
K., et al.
Hyperhomocysteinemia
and
low
pyridoxal
phosphate.
Common
and
independent
reversible
risk
factors
for
coronary
artery
disease.
Circulation.
1995
Nov
15
;92(10):2825-30.
1
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