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Transcript of Asbestos Prezi
What is Asbestos?
Asbestos was a popular building material used in the 19th and 20th century.
Lauded for its sound absorption, tensile strength, fire, chemical and electrical resistance as well as affordability.
Where is it found in everyday life?
At one point in time Asbestos was used in a vast variety of products from drywall to gas masks and brakepads to dental casts.
Dr. Montague Murry first noted the negative health effects in 1899!
First death accredited to asbestos occurred in 1906
As it became apparent that asbestos can be extremely dangerous to ones health, governments started to phase its use out or outright banned it
So why does this matter?
"A form of pneumoconiosis caused by inhalation of asbestos fibers which elicit potent inflammatory responses in the parenchyma of the lung" -ICD9
How does it affect the respiratory system?
There is no cure for asbestos only treatment to alleviate symptoms namely:
Medication to thin lung fluid
Cessation of smoking
Pneumococcal pneumonia and influenza immunizations.
Regular screenings for complications and secondary infections
Is there anything we can do?
- Slow onset dyspnea
- Chest pain
- Tightness in the chest
And only in some cases:
- Finger clubbing
- Nail abnormalities
- Jugular venous distention
- Hepatojugular reflux
- Pedal oedema
Scarring of the lung tissue will occur with the inhalation of asbestos fibers.
Amphibole (thin & straight) fibers reaching the alveoli illicit a inflammatory response.
Failed immunological responses lead
to fibrosis as connective tissue is formed around the asbestos fibers
Alveolar walls thicken, reducing elasticity
Prevention and safety procedures are key!
A. Burke -22149724
9/11 Destruction of the Twin Towers
Asbestos & SU 1 :
Reduces alveoli elasticity due to physiological damage
Oxygen uptake reduces due to less effective gas exchange
Work increases, due to decreased compliance
Asbestos & SU2
Ventilation perfusion ratio: lungs are scarred and is ‘smaller’ and therefore the lung can take less air
/ normal ventilation-perfusion ratio is (V/Q)= 0.8-1.2
Physiological dead space – impaired gas exchange take place and therefore ventilation is wasted
Asbestos & SU 3
The oxygen-hemoglobin dissociation curve moves right
Asbestos & SU 4
Decreased respiratory volume
Increase in stretch receptor firing
Increase in DRG inspiratory neuron firing
Increased ventilation rate
No concrete evidence
Alleman, J. & Massman, B., 1997. Asbestos Revisited. Scientific American, July, 277(1), pp. 70-75.
Mandal, A., 2013. News Medical. [Online]
Available at: http://www.news-medical.net/health/What-is-Dyspnea.aspx
[Accessed 8 April 2014].
National Institute of Environmental Health Sciences, 2005. Asbestos CAS No. 1332-21-4, Washington, D.C.: Department of Health and Human Services .
Stephenson, J., 2007. World Trade Center : preliminary observations on EPA's second program to address indoor contamination [Interview] (20 June 2007).
Varkey, B., Varkkey, A. & Mosenifar, Z., 2013. Medscape. [Online]
Available at: http://emedicine.medscape.com/article/295966-clinical
[Accessed 9 April 2014].
Decreased gas exchange
Increased blood PCO2
Increased CO2 receptors
Increase in heart rate
Increased inter pleural pressure
Damage of alveoli and lung tissue
Our own hypothesis!