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Pathologie Hemostase

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Ella Kamphuis

on 16 October 2012

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Transcript of Pathologie Hemostase

Pathologie Hemostase 1. vasoconstrictie
2. adhesie & aggregatie
3. fibrinevorming
4. fibrinolyse Trombocytopathie ziekte van
von Willebrand Pseudo-trombocytopenie Verhoogde afbraak / verbruik Verminderde aanmaak Erfelijk Verworven Primaire
hemostase Trombocytopenie Tekort: < 150 * 10^9 /L - Lab-artefact
- EDTA-fenomeen:
1 op 1000 patiënten - Auto-antistoffen
- Heparine geïnduceerde
- Ernstige bloedingen
- Diffuse intravasale stolling - Leukemie
- Medicatie
- Bestraling Ontbreken van of het niet functioneel zijn van receptoren op de trombocyten, voor adhesie of aggregatie. ziekte van
Bernard-Soulier ontbreken receptor adhesie ziekte van Glanzmann ontbreken receptor aggregatie - Medicatie
(ascal, aspirine of
- Auto-antistoffen - vWF onvoldoende aanwezig
- vWF onvoldoende functioneel Secundaire hemostase APTT: normaal
PT: verlengd APTT: verlengd
PT: normaal APTT: verlengd
PT: verlengd Medicatie Verbruik van stollingsfactoren Factor II, V, X of fibrinogeen tekort Medicatie
(cumarine) Vitamine K
tekort Factor VII
tekort Heparine Factor VIII, IX, XI, XII, PK, HK Remmer: lupus anticoagulans Vitamine K-tekort Leverfalen
Full transcript